罪魁祸首是在采矿过程中释放的可吸入灰尘,该矿物含有煤和其他足够小的矿物颗粒,可以深入呼吸肺部,在那里他们可以阻挡空气通道并引起肺组织的瘢痕。
CWP有两个阶段,简单而复杂。在简单的舞台上,小结节在肺部发展。在这个阶段,受影响的工人可能无症状,或呼吸急促和慢性咳嗽。
如果工人继续暴露于高浓度的可吸入煤尘,疾病会导致慢性气流限制和对复杂的CWP进行进展。这导致肺中大,致密的肌瘤,显着降低肺功能并且可能是致命的。
因为没有治愈,拯救来自CWP人士的唯一方法是防止他们首先呼吸危险的可吸入煤尘。
This prevention is the aim of a joint research initiative between The University of Queensland, the Queensland Government’s Safety in Mines Testing and Research Station (Simtars) and New South Wales-based industry organisation Coal Services, with funding from the Australian Coal Association Research Program (ACARP).
The ‘Improving respirable coal dust exposure monitoring and control’ project, led by Professor David Cliff from UQ’s Minerals Industry Safety and Health Centre (SMI-MISHC), has two main aims: to establish the best ways of measuring mine workers’ exposure to respirable dusts, and then using these measurements to test the effectiveness of different exposure reduction methods.
Nikky Labranche是主要的采矿工程师表示,该项目旨在改善昆士兰州矿工安全安全的核心。